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The observed selective reduction in TEM, T c -EM, T h -EM with rLcn2 treatment suggests a potential dampening of immune memory, indicating that rLcn2 may help modulate long-term immune responses

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This activation subsequently cleaves and matures inflammatory factors, including IL-1β and IL-18, and can also activate pyroptosis and other pathways of inflammatory cell death, serving as a critical regulatory hub in the body’s innate immune response and inflammatory reaction [179]