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This model involves administration of mice with DSS in drinking water for a period of 8–10 days to induce colonic epithelial barrier disruption and ultimately lead to colitis (Baydi et al

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Within the GHSR1a-MC3R dimer, the GHSR1a inverse agonist SP-analog eliminates amplification of MC3R signaling (Rediger et al., 2011), an effect likely due to the dependence on GHSR1a constitutive activity