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Although these levels remained below those observed in juvenile animals, T3-induced proliferation recapitulated developmental susceptibility, definitively linking replication stress, not mere adduct accumulation, to downstream DNA damage and mutagenesis

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As predicted, our experiments showed that conditioned medium derived from CAV-1-silenced keratinocytes promotes HUVEC tube formation (although the effect did not reach statistical significance) and that the restoration of CAV-1 activity in keratinocytes significantly reduced the HUVEC tube formation induced by the conditioned medium