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DHODH activity is essential for proliferating cells owing to their increased demand for nucleotides, and DHODH inhibition has long been studied as a therapeutic strategy in autoimmune diseases (for example, via leflunomide) and cancer, where it can lead to cell cycle arrest, DNA damage and replication stress 42,44,45

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engineered glioblastoma-targeting nanoparticles by conjugating matrix metalloproteinase-2 (MMP-2)-responsive HA-PLGLAG-dEGCG polymers with anti-B7-H3 × CD3 bispecific antibodies [51]