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Despite compelling preclinical evidence, clinical translation of Nrf2 activators for MIRI remains in its infancy, with a significant lack of published trial data specifically in this acute setting (summarized in Table 1)
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Indeed, several additional metabolic targets of p53 also contribute to its ferroptotic response and the defects in p53-mediated ferroptosis caused by an African-specific polymorphism in the TP53 gene impair its tumor suppressor function [[420], [421], [422], [423]]