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Once selected, transfected cell lines derived from the CHO DHFR negative host do not require MTX in the culture medium

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Inhibition of non-c-Met targets may enhance the antitumor activity of non-selective c-Met inhibitors, but is also correlated with increased toxicity and limits the dose so that inhibitors cannot effectively suppress c-Met ( MicroRNAs Fortunately, using potential miRNAs for suppressing aberrant c-Met signal is an emerging and promising therapy strategy that bypasses traditional approaches (Karagonlar et al., 2015) (Table 2)