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Moreover, the immunogenicity response of GX-G3 was lower in monkeys than in rats, which was relevant to show less inhibition of toxicokinetic profiles in monkeys, at least 1 mg/kg administrated group, compared to rats

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This disrupts the hair follicle immune privilege (IP), upregulating major histocompatibility complex (MHC) class I and II antigens expression and culminating in autoimmune attack [15, 16, 17]