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PMC Part III: The Synergistic Combination — Mechanistic Rationale Complementary Angiogenic Inputs Both compounds independently promote new blood vessel formation through mechanistically distinct pathways: BPC-157 via VEGFR2 receptor signaling and Akt-eNOS activation, and TB-500 via actin cytoskeleton regulation of endothelial cell migration

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Studies show diminishing returns and potential harm from multiple injections into the same joint