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Although cells can withstand low levels of ROS, processes within mitochondria ensure that levels do not increase to unfavorable amounts, all without compromising the contribution of ROS to signaling pathways

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In addition, the hepatic oxidative stress caused by MTX, as indicated by the increased production of MPO, elevated protein carbonylation, lipid peroxidation, and reduced activity of anti-oxidant enzymes, was alleviated by prior administration of EGCG