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In cell culture systems, the upregulation of these heat shock proteins similarly caused reductive stress with increased GSH and decreased ROS [48, 49]

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Given the previously discussed importance of the non-canonical inflammasome in sepsis and controling intracellular Gram-negative bacterial pathogens , these findings suggest that HO-1 limits the activation of non-canonical inflammasome signalling by modulating heme levels (Kajiwara et al., 2014