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FUS suppresses G 4 C 2 repeat-induced toxicity via its RNA-binding activity We next investigated whether the suppressive effects of FUS on G 4 C 2 repeat-induced toxicity are mediated by its binding to G 4 C 2 repeat RNA, using flies expressing FUS with mutations in the RRM ( FUS-RRMmut ), which have been reported to eliminate its RNA-binding activity (Daigle et al., 2013)

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Supplementation with antioxidants in the treatment of graves' disease: the effect on the extracellular antioxidative parameters