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This review systematically summarizes the molecular mechanisms underlying the dynamic equilibrium of cellular redox state, including the intracellular sources of ROS and the multilayered antioxidant defense network

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Research indicates that gut microbial metabolites, such as SCFAs and α-tocopherol, can activate various G protein-coupled receptors (e.g., FFAR2, OLF78, GPBAR1) on the surface of EC cells, thereby promoting the expression of tryptophan hydroxylase 1 (TPH1) and the release of 5-HT (252–255)