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Due to the high reactivity of Prx and Gpx (and abundance in the case of Prx), direct oxidation of GSH and other important redox-regulated proteins (like GAPDH or protein tyrosine phosphatase) are prevented up till the concentration of H 2 O 2 reaches tens of μ M -s, and hence, the redox signal that involves H 2 O 2 is most likely transmitted via Prx and/or Gpx

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Remote postischemic conditioning promotes stroke recovery by shifting circulating monocytes to CCR2+ proinflammatory subset