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Some of these interactions involve the upregulation of CD38, mediated by adipocyte-secreted angiotensin II (Ang II), which has been identified as the direct cause of increased expression of acetyl-CoA synthetase (ACSS2) in MM plasma cells, promoting growth by stabilizing the oncoprotein IRF4 in vitro and in vivo (95)
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AOD-9604 versus cagrilintide / tirzepatide (GLP-1 co-agonists) GLP-1 analogues (semaglutide, tirzepatide, cagrilintide) constitute the major pharmacological class for obesity in 2024-2025, with clinically documented weight reductions of 15 to 25% over 68 weeks (STEP, SURMOUNT trials)
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